CONSCIOUS COMBINING EXPLAINED: THE LOW-CAL EXPOSE: EGGS, SKINLESS CHICKEN BREAST, RAW VEGETABLES AND MELONS
Eggs. These are a damned if you do, damned if you don't item. The ingredient that makes eggs digestible, the lecithin, is in the yolk and is destroyed when you cook them. On the other hand, there is an ingredient in the egg white which when eaten raw destroys B vitamins. The latest craze is to eat only the cooked whites—well, whatever turns you on. I was always a yolk dunker.
Skinless Chicken Breast (and other denuded animal proteins). The skin contains essential nutrients. When you strip it away, you are adversely affecting the digestibility of the fowl. Besides, doesn't it taste better the way nature made it—with the skin on?
Raw Vegetables. Yes, they are low-calorie, but they are low-energy too. So why bother?
Melons. With the important exception of watermelon, which has marvelous enzymatic powers, most melons have no fiber and no digestive enzymes. They are not only hard to digest, but they are bloaters as well.
Centrophenoxine is a smart drug and a brain antiaging substance based on the molecular formula of the smart nutrient DMAE, commonly found in seafood and nutritional supplements. Like DMAE, centrophenoxine can supply the building blocks for the synthesis of the neurotransmitter acetylcholine. In addition, it can remove lipofuscin deposits in the brain, heart, and skin. Studies have shown that the removal of lipofuscin deposits in the brain is correlated with improved mental functioning, while increased lipofuscin deposits are associated with decreased cognitive abilities.
Centrophenoxine, like DMAE, facilitates nerve transmission across synapses, the tiny spaces between neurons where neurotransmitters are released from one nerve cell to communicate with other nerve cells. Some research indicates that centrophenoxine is as effective as DMAE in neutralizing free radicals. Other research suggests that centrophenoxine is more effective than DMAE in slowing down the accumulation of lipofuscin deposits, at least in experiments on laboratory animals.
PRECAUTIONS: People who are hyperactive, easily excitable, have severe hypertension (high blood pressure), or suffer from or are susceptible to convulsions or involuntary musculoskeletal movements should avoid using centrophenoxine. Nursing mothers should also avoid using this smart drug because it can appear in breast milk. Side effects include insomnia, motion sickness, muscular tremors, hyperexcited states, and even a paradoxical drowsiness. Some instances of depression have also been reported. Centrophenoxine is more likely to cause insomnia if taken late in the day.
Dosage commonly used: 1,000 to 3,000 mg per day.
The first day my son stayed at school until three in the afternoon, he had to take lunch with him. Since I wanted this to be a wonderful experience and to reinforce how great it was to stay all day, I prepared him his favorite lunch. Not only did I run out to the fast-food chicken store for the precise part of the chicken's anatomy he prefers, but I drove another mile and a half to a bakery where they make the kind of chocolate cupcakes he likes. I also threw in a small package of Doritos, one of those cute packages of fruit juice, and a napkin with Snoopy on it.
Let's look at what I did. First of all, the nutritional value of that meal stinks. Second, I wasted a lot of time, trouble, and money. But worst of all, I sent out a psychological message that said, "If you stay at school until three, you will be rewarded with food. »(I stood in the corner and punished myself when I realized what I had done.)
So we have been called to be soldiers in a battlefield. Your assignment, should you decide to accept it, is to fight your desire for food by being alert. Keep in mind that the very time of day when we finally let down and relax and want to reward ourselves with food for making it through such a hard day is the very time that we are to be in the watchtower, alert to the enemy. The times that we celebrate with food because something wonderful has happened are the times we actually must be very watchful, sober, and on our guard. The times when we seem bored, with nothing to do, when eating crosses our minds as a way to fill the day, are the very times that we must be most active and busy carrying out God's will and not our own. The time when we finally think everyone has gone to bed and no one is looking is the very time the room might be full of demons ready to lie to and torment us; but it is also filled with a great crowd of heavenly beings cheering us on to victory. And we are not even aware of it! I have spent most of my life asleep to the spiritual realm. "Wake up, O sleeper!"
We know that we are children of God, and that the whole world is under the control of the evil one.
Be self-controlled and alert. Your enemy the devil prowls around like a roaring lion looking for someone to devour. Resist him, standing firm in the faith, because you know that your brothers throughout the world are undergoing the same kind of sufferings.
And the God of all grace, who called you to his eternal glory in Christ, after you have suffered a little while, will himself restore you and make you strong, firm and steadfast. To him be the power for ever and ever. Amen.
We are warned many times to be alert. I am convinced that every one of us is here to learn to raise our consciousness level to be alert to God every hour of the day. That is why your battles will be something different every day and at different hours. You are not going to have the same exams. Yet, Satan is good at making us believe that we are OK and that there are no battles. Or, on the other end of the spectrum, the "accuser of the brethren" makes us feel as though God does not love us. Not true! His tactics are to remain in the background and chip away at us, or have us lose the battles so subtly that we do not even know that we are in a battle— much less losing it—like a boat slowly drifting to the sea. His major goal is to distance you from God and from truth.
In the early 1980s singer Karen Carpenter died of anorexia nervosa, and the media began to focus attention on this growing problem among American women. Today eating disorders—anorexia nervosa, bulimia nervosa, food addiction, and binge eating disorder—have reached alarming proportions in all segments of the population. To what can we attribute this epidemic, and, more important, what can we do about it?
The reasons for eating disorders are not clear, in part because research in this field is new—less than two decades old—and in part because of the way investigations have been conducted in the past. Kelly Brownell, Ph.D., director of the Yale Center for Eating and Weight Disorders and coauthor of Eating Disorders and Obesity: A Comprehensive Handbook, notes that up until recently, eating disorder researchers have worked apart from scientists investigating obesity, and the two groups have not communicated. Brownell explains, "The issue of what governs hunger and satiety, for example, would he very important to know in the eating disorders field, but almost all of the study on that goes on in die obesity field. There's a lot of important work on body image that goes on in the eating disorders field, and the obesity field doesn't study it very much."
Fortunately, more sharing is starting to take place, and our understanding of these issues is becoming enriched. Current thinking attributes eating disorders to a combination of physiological, psychological, and sociological factors. There is also a genetic component: New re-search—specifically, investigations of identical twins raised apart—supports the hypothesis that eating disorders are, to an extent, inherited. While such studies may seem to take the emphasis off cultural factors, our culture obviously plays a large role in the development of eating disorders, as evidenced by the increasing incidences of anorexia and bulimia in the past ten years.
Ed, a 35-year-old government employee, came to me for help when his weight reached 275 pounds. He had little energy or stamina and suffered from shortness of breath. He had high blood pressure, high cholesterol, and realized that his eating was totally out of control. He reported a history of binge eating that went back several years.
One of Ed's eating problems was that he never felt full. Here is how he described it.
I seem to be hungry most of the time. When I eat a meal, I usually eat very rapidly. I guess that's because I'm so hungry. I usually eat a lot. I've gotten used to very large portions. But, once the meal is over, I really don't feel full. This doesn't make sense since I usually eat about twice as much as I should.
The problem that Ed has with not knowing when he is full is an important one that must be resolved. By eating rapidly, Ed is making the problem worse since it takes about 20 minutes after eating for hunger messages to be turned off by your brain. One of the key concepts in my treatment program is to make you aware of satiety (the feeling of stomach fullness) as well as real physical hunger. Ed learned to slow his eating and began to pay attention to physical sensations of fullness during and after his meal. I am pleased to say that he lost 85 pounds and has conquered his binge-eating problem.
Your uncle, who's pushing 65, smokes like a belching mill, pigs out on double omelettes, oily parathas, mutton chops and chicken biryanis, is addicted to his double round of chhota pegs, and for exercise nods off regularly before DD II. Yet, there he is, surviving — and thriving.
You can't help but contrast his fortunes with the luckless destiny of the 20-something footballer who clutched his chest and fell dead on the field in the middle of a game.
Apart from the fact that there's no justice in the world, is there anything more rational to explain your uncle's insouciant grip on life while a young, clear-eyed, fit-looking youth is felled dead out of the blue? There well may be. Some years ago, researchers at the Bowman Gray School of Medicine in the U.S. identified a common genetic mutation that makes people resistant to dietary cholesterol, allowing them to eat even 1000 mg. a day without raising the level of cholesterol in their blood.
On the other hand some people, because of their genes again, inherit a disorder called hypercholesterolemia due to which their bodies are unable to efficiently dispose of excess blood cholesterol, setting them up for heart disease. Researchers have managed to identify one gene (known as apo E4) which increases the tendency for those who have it to also have high cholesterol levels and a greater risk of heart disease.
But it's not only our genes that throw the switches that decide the fate of our hearts. In fact, there are far too many pieces still missing in the jigsaw puzzle that adds up to heart disease - and eventually a heart attack. The more we learn, the more we realize how much more we need to learn. Two decades ago, a leading cardiac researcher in the United States predicted that in a few years we'd have the problem of heart disease licked. An over-optimistic prophesy, of course; heart disease today afflicts more numbers of people worldwide than ever before.
The good news is that we are today well enough informed to significantly cut our risks of getting heart disease. We also have better drugs to prevent heart disease and better drugs to treat attacks.